Slow-wave sleep, diabetes, and the sympathetic nervous system
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چکیده
منابع مشابه
Loss of slow-wave sleep.
A 40-year-old Hispanic American male, who weighed 165 lb and was 5’11” tall, consented to participate in a 3-day sleep protocol and was admitted to the UCLA General Clinical Research Center. The patient complained of fatigue during the day and difficulty initiating sleep, along with 3 to 4 awakenings per night. He denied snoring and having any difficulty breathing during sleep. A 2-week sleep d...
متن کاملSympathetic nervous system neuroplasticity.
In human and experimental models of heart failure there is widespread change in the sympathetic nervous system. This involves preferential activation of the sympathetic nervous outflow to the heart, impairment of norepinephrine reuptake by sympathetic nerves, and reduction of the content of norepinephrine in the failing myocardium, attributable in part to sympathetic neuronal rarefaction.1–4 Mi...
متن کاملSympathetic nervous system and hypertension.
Sympathetic Nervous System and Hypertension Print ISSN: 0194-911X. Online ISSN: 1524-4563 Copyright © 2013 American Heart Association, Inc. All rights reserved. is published by the American Heart Association, 7272 Greenville Avenue, Dallas, TX 75231 Hypertension doi: 10.1161/HYPERTENSIONAHA.111.00633 2013;61:556-560; originally published online January 28, 2013; Hypertension. http://hyper.ahajo...
متن کاملRelationship of arousals from sleep to sympathetic nervous system activity and BP in obstructive sleep apnea.
STUDY OBJECTIVE Obstructive sleep apnea (OSA) patients have a high frequency of arousals. We hypothesized that arousals significantly influence tonic sympathetic nervous system function. DESIGN We examined the association of 11 variables measuring sympathetic activity, including plasma norepinephrine (NE), urinary NE, and BP measurements, with movement and cortical arousals. PATIENTS Sixty-...
متن کاملThe sympathetic nervous system and heart failure.
Heart failure (HF) is a syndrome characterized by upregulation of the sympathetic nervous system and abnormal responsiveness of the parasympathetic nervous system. Studies in the 1980s and 1990s demonstrated that inhibition of the renin-angiotensin-aldosterone system with angiotensin-converting enzyme inhibitors improved symptoms and mortality in HF resulting from systolic dysfunction, thus pro...
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ژورنال
عنوان ژورنال: Proceedings of the National Academy of Sciences
سال: 2008
ISSN: 0027-8424,1091-6490
DOI: 10.1073/pnas.0711635105